Search results for "Silent mutation"

showing 6 items of 6 documents

A Network Model for the Correlation between Epistasis and Genomic Complexity

2008

The study of genetic interactions (epistasis) is central to the understanding of genome organization and evolution. A general correlation between epistasis and genomic complexity has been recently shown, such that in simpler genomes epistasis is antagonistic on average (mutational effects tend to cancel each other out), whereas a transition towards synergistic epistasis occurs in more complex genomes (mutational effects strengthen each other). Here, we use a simple network model to identify basic features explaining this correlation. We show that, in small networks with multifunctional nodes, lack of redundancy, and absence of alternative pathways, epistasis is antagonistic on average. In c…

0106 biological sciencesSilent mutationGenome evolutionDNA Mutational Analysislcsh:MedicineBiology010603 evolutionary biology01 natural sciencesGenomeModels BiologicalCorrelation03 medical and health sciencesComputational Biology/Metabolic NetworksGenetics and Genomics/Population GeneticsAnimalsHumanslcsh:Science030304 developmental biologyGenomic organization0303 health sciencesEvolutionary BiologyMultidisciplinaryComputational Biology/Systems BiologyGenomeEvolutionary Biology/Evolutionary and Comparative GeneticsModels GeneticHuman evolutionary geneticsSystems Biologylcsh:RRobustness (evolution)Computational BiologyGenetics and GenomicsEpistasis GeneticGenomicsModels TheoreticalEvolutionary biologyMutationEpistasislcsh:QAlgorithmsResearch ArticlePLoS ONE
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Systematic screening for mutations in the promoter and the coding region of the 5-HT1A gene.

1995

In the present study we sought to identify genetic variation in the 5-HT{sub 1A} receptor gene which through alteration of protein function or level of expression might contribute to the genetic predisposition to neuropsychiatric diseases. Genomic DNA samples from 159 unrelated subjects (including 45 schizophrenic, 46 bipolar affective, and 43 patients with Tourette`s syndrome, as well as 25 healthy controls) were investigated by single-strand conformation analysis. Overlapping PCR (polymerase chain reaction) fragments covered the whole coding sequence as well as the 5{prime} untranslated region of the 5-HT{sub 1A} gene. The region upstream to the coding sequence we investigated contains a …

GeneticsSilent mutationMutationBipolar DisorderBase SequenceMolecular Sequence DataNucleic acid sequenceBiologyGene mutationmedicine.disease_causeReceptors SerotoninGenetic variationMutationGenetic predispositionmedicineSchizophreniaCoding regionHumansGeneReceptors Serotonin 5-HT1Genetics (clinical)Polymorphism Single-Stranded ConformationalTourette SyndromeAmerican journal of medical genetics
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Simultaneous identification of campylobacters and prediction of quinolone resistance by comparative sequence analysis.

1997

Comparative sequence analysis of a 30-bp segment in the quinolone resistance-determining region of campylobacters not only allows for the detection of base changes associated with resistance but also is a powerful tool for species identification based on silent mutations.

Microbiology (medical)Silent mutationDNA Bacterialmedicine.drug_classSequence analysisSequence alignmentBiologymedicine.disease_causeDNA gyrasePolymerase Chain Reactionlaw.inventionAnti-Infective AgentsSpecies SpecificitylawmedicinePolymerase chain reactionAntibacterial agentGeneticsMutation4-QuinolonesSequence Homology Amino AcidCampylobacterDrug Resistance MicrobialSequence Analysis DNAQuinoloneDNA Topoisomerases Type IIDNA GyraseSequence AlignmentResearch Article
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Missense and nonsense mutations in melanocortin 1 receptor (MC1R) gene of different goat breeds: association with red and black coat colour phenotype…

2009

Abstract Background Agouti and Extension loci control the relative amount of eumelanin and pheomelanin production in melanocytes that, in turn, affects pigmentation of skin and hair. The Extension locus encodes the melanocortin 1 receptor (MC1R) whose permanent activation, caused by functional mutations, results in black coat colour, whereas other inactivating mutations cause red coat colour in different mammals. Results The whole coding region of the MC1R gene was sequenced in goats of six different breeds showing different coat colours (Girgentana, white cream with usually small red spots in the face; Maltese, white with black cheeks and ears; Derivata di Siria, solid red; Murciano-Granad…

Silent mutationCoatlcsh:QH426-470GenotypeMolecular Sequence DataNonsense mutationPopulationMutation MissenseMELANISMBiologyPolymorphism Single NucleotideAGOUTI PROTEINSettore AGR/17 - Zootecnica Generale E Miglioramento GeneticoMSH RECEPTORBREEDSMC1RGeneticsAnimalsMissense mutationGenetics(clinical)Amino Acid Sequencecoat colour; MC1R; goatAlleleHair ColoreducationAllele frequencyPOPULATIONPOLYMORPHISMSAllelesGenetics (clinical)Geneticseducation.field_of_studySTIMULATING-HORMONE-RECEPTORGoatsgoatCATTLE BREEDSSequence Analysis DNAMolecular biologyCOAT COLORlcsh:GeneticsPhenotypeCodon NonsensePIGMENTATIONWHITEReceptor Melanocortin Type 1EXTENSIONcoat colourResearch ArticleMelanocortin 1 receptorBMC Genetics
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Systematic screening for mutations in the human serotonin-2A (5-HT2A) receptor gene: Identification of two naturally occurring receptor variants and …

1996

A statistically significant association between a silent mutation (102T/C) in the serotonin-2A (5-HT2A) receptor gene and schizophrenia has recently been reported in a sample of Japanese patients and healthy controls. This finding suggests that genetic predisposition to schizophrenia may be affected by a functional 5-HT2A receptor variant that is in linkage disequilibrium with 102T/C. In the present study, we have sought to identify genetic variation in the 5-HT2A receptor gene by screening genomic DNA samples from 91 unrelated subjects comprising 45 patients with schizophrenia and 46 healthy controls by using single-strand conformation analysis. We have identified four nucleotide sequence …

Silent mutationLinkage disequilibriumMolecular Sequence DataRestriction MappingBiologymedicine.disease_causePolymerase Chain ReactionReference ValuesGenetic variationConfidence IntervalsGeneticsGenetic predispositionmedicineHumansPoint MutationReceptor Serotonin 5-HT2AAmino Acid SequenceAlleleAllele frequencyAllelesGenetics (clinical)DNA PrimersGenetic associationGeneticsMutationPolymorphism GeneticBase SequenceChromosomes Human Pair 13Chromosome MappingGenetic VariationExonsReceptors SerotoninSchizophreniaHuman Genetics
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Quasispecies dynamics and fixation of a synonymous mutation in hantavirus transmission.

2008

RNA-dependent RNA polymerases, the key enzymes in replication of RNA viruses, have a low fidelity; thus, these viruses replicate as a swarm of mutants termed viral quasispecies. Constant generation of new mutations allows RNA viruses to adapt swiftly to a novel environment through selection of both pre-existing and de novo-generated genetic variants. Here, quasispecies dynamics were studied in vivo in controlled hantavirus transmission from experimentally infected to naïve rodents through infested cage bedding. An elementary step of virus microevolution was apparent, as one synonymous mutation (A759G) repeatedly became fixed in the viral RNA quasispecies populations in the recipient animals.

Silent mutationOrthohantavirusvirusesHantavirus InfectionsViral quasispeciesBiologyVirusEvolution Molecular03 medical and health sciencesVirologyAnimalsPolymerase030304 developmental biologyHantavirusGenetics0303 health sciences030306 microbiologyArvicolinaeRNASequence Analysis DNAbiology.organism_classificationVirology3. Good healthViral evolutionMutationbiology.proteinRNA ViralBunyaviridaeThe Journal of general virology
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